Medical Pharmacology Question Bank

Chapter 15: Local Anesthetic Pharmacology — Module 5: Neuraxial Anesthesia
Clinical Vignette (11 questions)


1. A 31-year-old woman at 39 weeks' gestation receives a spinal anesthetic for elective cesarean delivery and is positioned supine on the operating table. Within three minutes her systolic blood pressure falls from 118 to 72 mmHg, she reports nausea, and the fetal heart rate tracing shows a prolonged deceleration to 90 beats per minute. What is the most appropriate immediate action?

  • A) Place the patient in steep reverse Trendelenburg and withhold vasopressors until the pressure falls further
  • B) Apply left uterine displacement (tilt or right-hip wedge) and give a phenylephrine bolus, treating aortocaval compression compounded by spinal sympathectomy
  • C) Administer intravenous labetalol to control the maternal hemodynamics
  • D) Proceed immediately to general anesthesia and intubation as the first maneuver
  • E) Give a large dose of intravenous furosemide to offload the circulation

ANSWER: B

Rationale:

In a term parturient placed supine, the gravid uterus compresses the inferior vena cava and aorta, and this aortocaval compression compounds the sympathectomy-induced vasodilation of the spinal to produce rapid maternal hypotension that preferentially reduces uteroplacental flow, manifesting as the fetal deceleration. The correct immediate response is to relieve the mechanical compression with left uterine displacement and to restore maternal pressure and uteroplacental perfusion with phenylephrine, the preferred vasopressor in this setting.

  • Option A: Option A worsens the situation by delaying treatment and using a position that does not relieve aortocaval compression.
  • Option C: Option C gives a beta-blocking antihypertensive to a hypotensive patient, which would deepen the hypotension.
  • Option D: Option D escalates to general anesthesia before attempting the simple, rapidly effective maneuvers that usually correct the problem.
  • Option E: Option E removes intravascular volume with a diuretic, aggravating the hypotension.

2. A 54-year-old man receives a spinal anesthetic for an inguinal hernia repair. Over several minutes he becomes anxious and dyspneic, reports tingling in both hands, and develops a heart rate of 44 beats per minute with a systolic pressure of 80 mmHg; the sensory level is testing at approximately T2. Which response best addresses the underlying problem?

  • A) Reassure the patient that the symptoms are anxiety, give a benzodiazepine, and continue without hemodynamic support
  • B) Administer intravenous esmolol to slow what appears to be a stress-related tachycardia
  • C) Give a vasodilator to improve coronary perfusion during the episode
  • D) Recognize an evolving high spinal: support oxygenation and ventilation (supplemental oxygen, with intubation if respiratory failure develops), give rapid intravenous fluids, and treat the bradycardia-with-hypotension using ephedrine for combined alpha and beta support plus atropine for the vagally mediated bradycardia
  • E) Sit the patient fully upright immediately to lower the block, then leave him unmonitored

ANSWER: D

Rationale:

The ascending sensory level near T2, hand tingling, dyspnea, bradycardia, and hypotension together indicate a high spinal: the block has reached the cardiac accelerator fibers (T1 to T4), removing sympathetic drive to the heart, while threatening the muscles of respiration. Management is simultaneous airway and breathing support, rapid fluids, and ephedrine (combined alpha and beta activity) for hypotension accompanied by bradycardia, with atropine for the vagally mediated slowing. Option B gives a beta-blocker to a bradycardic, hypotensive patient, worsening both. Option C uses a vasodilator that would deepen the hypotension. Option E relies on abrupt repositioning of an already-set block and dangerously leaves a deteriorating patient unmonitored.

  • Option A: Option A misattributes a dangerous physiologic event to anxiety and withholds needed support.

3. A laboring patient with a dilute labor epidural infusion (low-concentration bupivacaine with fentanyl) has had excellent analgesia for hours. As the fetal head descends in late second stage, she develops new intense perineal pressure and pain, although her abdominal contraction pain remains well controlled. The catheter aspirates negative and was previously functioning well. What is the most appropriate next step?

  • A) Recognize incomplete sacral root coverage: administer a more concentrated local anesthetic bolus through the catheter and/or reposition the patient upright for a period to direct solution toward the sacral nerve roots
  • B) Conclude the catheter has failed and remove it, then begin systemic opioids alone
  • C) Diagnose an epidural hematoma and obtain emergent MRI
  • D) Give a large intravenous fluid bolus, since perineal pain in labor reflects hypovolemia
  • E) Convert immediately to general anesthesia for delivery

ANSWER: A

Rationale:

This is a single-patient reasoning problem about sacral coverage. Late second-stage perineal pain with preserved abdominal analgesia is the classic picture of inadequately blocked sacral nerve roots, which are larger and harder to reach and are often incompletely covered by a dilute infusion in a recumbent patient. The remedy applies the concentration-density relationship and sacral anatomy: give a more concentrated bolus and/or reposition the patient upright to direct solution caudally toward the sacral roots. Option B discards a functioning catheter and abandons effective regional analgesia for an inferior systemic approach. Option C invokes a catastrophic complication that does not fit a comfortable patient with isolated perineal pain and a previously functioning catheter. Option E escalates to general anesthesia when a simple dosing and positioning adjustment is indicated.

  • Option D: Option D misattributes perineal labor pain to hypovolemia.

4. A laboring patient with a well-functioning labor epidural develops a category III fetal heart rate tracing, and the obstetric team calls for an emergent cesarean delivery within minutes. You elect to extend the existing epidural to surgical anesthesia rather than perform a spinal or induce general anesthesia. Which agent injected through the catheter provides the fastest, most reliable dense surgical block?

  • A) Additional 0.0625% bupivacaine, matching the labor infusion concentration
  • B) Intrathecal morphine injected through the epidural catheter
  • C) 3% chloroprocaine, whose high concentration drives a large mass of drug across the nerve membranes to produce a dense surgical block within roughly 6 to 10 minutes
  • D) A dilute ropivacaine 0.1% bolus
  • E) An intravenous lidocaine infusion in place of any epidural top-up

ANSWER: C

Rationale:

When an existing epidural must be extended to surgical anesthesia under time pressure, 3% chloroprocaine gives the fastest, most reliable onset, on the order of 6 to 10 minutes. Although chloroprocaine has an unfavorable pKa, its very high concentration delivers a large mass of drug that crosses the nerve membranes by mass action, overcoming the ionization disadvantage to produce a dense block quickly.

  • Option A: Option A keeps an analgesic concentration that cannot achieve surgical density in time.
  • Option B: Option B uses a slow-onset neuraxial opioid that does not establish operative anesthesia within minutes.
  • Option D: Option D is another analgesic-concentration solution unsuited to rapid surgical block.
  • Option E: Option E substitutes a systemic infusion that does not provide surgical anesthesia of the operative field.

5. A healthy 35-year-old is scheduled for an elective outpatient knee arthroscopy expected to last about 40 minutes, with planned same-day discharge. The patient prefers a spinal anesthetic. Which agent best matches this clinical goal?

  • A) Hyperbaric lidocaine 5%, accepting its known rate of transient neurologic symptoms for the sake of speed
  • B) Hyperbaric bupivacaine 0.5% at a full surgical dose, accepting a 90 to 150 minute block
  • C) Intrathecal morphine as the sole agent to provide the operative block
  • D) Tetracaine with epinephrine to deliberately prolong the block
  • E) Preservative-free chloroprocaine, which provides rapid onset, a short and predictable duration suited to a brief case, prompt recovery for same-day discharge, and minimal risk of transient neurologic symptoms

ANSWER: E

Rationale:

The decision turns on matching agent duration and recovery profile to a short ambulatory case with same-day discharge. Preservative-free chloroprocaine offers rapid onset, a short predictable duration, full recovery suited to prompt discharge, and a very low rate of transient neurologic symptoms, making it the best fit.

  • Option A: Option A selects an agent with a recognized rate of transient neurologic symptoms when a low-risk alternative exists.
  • Option B: Option B uses a long-acting agent whose 90 to 150 minute block would needlessly delay discharge after a 40-minute case.
  • Option C: Option C misuses intrathecal morphine, an analgesic adjunct, as if it were a surgical anesthetic, and it would not provide the operative block while carrying delayed respiratory risk.
  • Option D: Option D deliberately prolongs the block, the opposite of the ambulatory goal.

6. A patient is three days into a continuous thoracic epidural infusion after major abdominal surgery. Over the preceding day he developed a fever to 38.7 degrees C, increasing back pain localized to the catheter insertion site, and a rising white blood cell count; he now reports new lower-extremity weakness. What is the most likely diagnosis and the correct management?

  • A) Pharmacologic over-blockade from the infusion; simply reduce the rate and reassess in the morning
  • B) Epidural abscess: obtain urgent spine MRI, arrange emergent neurosurgical decompression, and begin pathogen-directed antibiotics after appropriate cultures, recognizing Staphylococcus aureus as the most common organism
  • C) Post-dural puncture headache evolving into back pain; arrange an epidural blood patch
  • D) A simple superficial site infection; continue the infusion and apply a warm compress
  • E) Expected postoperative deconditioning; encourage early ambulation without imaging

ANSWER: B

Rationale:

The subacute course over days with fever, insertion-site back pain, leukocytosis, and then neurologic deterioration is the characteristic presentation of an epidural abscess, most often due to Staphylococcus aureus. Because neurologic deficit is present, the correct management is urgent spine MRI, emergent neurosurgical decompression, and pathogen-directed antibiotics after cultures; antibiotics alone are insufficient once a deficit has developed. Option A dangerously dismisses a febrile, leukocytotic patient with a new deficit as simple over-blockade. Option C misassigns the picture to post-dural puncture headache, which is a postural headache, not a febrile progressive myelopathy. Option D trivializes a deep neuraxial infection with neurologic involvement as a superficial site issue.

  • Option E: Option E attributes a red-flag neurologic deficit to deconditioning and omits the imaging and decompression the situation demands.

7. A postoperative patient with a thoracic epidural catheter is receiving prophylactic low-molecular-weight heparin (LMWH). The surgical team asks you to remove the epidural catheter so the patient can be discharged. The last LMWH dose was given a few hours ago, and another dose is due soon. What is the correct approach to catheter removal?

  • A) Time the removal to observe the recommended interval after the last LMWH dose and before the next dose, because catheter removal disrupts epidural vessels and carries a hematoma risk comparable to insertion, then hold the subsequent dose for the recommended interval after removal
  • B) Remove the catheter immediately regardless of LMWH timing, since removal cannot cause an epidural hematoma
  • C) Remove the catheter only after permanently discontinuing all anticoagulation for the remainder of the admission
  • D) Time only the insertion of catheters to anticoagulant dosing; removal timing is irrelevant
  • E) Give an extra dose of LMWH immediately before removal to ensure smooth withdrawal

ANSWER: A

Rationale:

The governing principle is that neuraxial anticoagulation intervals apply to catheter removal just as to placement, because withdrawing the catheter disturbs epidural vessels and carries comparable hematoma risk. With prophylactic LMWH, the catheter should be removed only after the recommended interval has elapsed since the last dose, and the next dose should be delayed for the recommended interval after removal. Option B is dangerously wrong because removal absolutely can precipitate an epidural hematoma. Option C is unnecessarily extreme; timed removal does not require abandoning all anticoagulation. Option E is hazardous because dosing LMWH immediately around removal maximizes, rather than minimizes, hematoma risk.

  • Option D: Option D ignores the well-established removal-timing requirement.

8. Two days after a labor epidural complicated by a recognized dural puncture, a patient has a severe headache that is mild when she lies flat and becomes disabling within minutes of sitting upright, with associated neck stiffness and photophobia. Bed rest, oral hydration, and caffeine over the past 24 hours have not provided meaningful relief, and she is unable to care for her newborn. What is the most appropriate definitive treatment?

  • A) Continue conservative measures unchanged for another full week before considering anything further
  • B) Begin broad-spectrum intravenous antibiotics for presumed meningitis
  • C) Perform a repeat lumbar puncture to drain cerebrospinal fluid and lower the pressure
  • D) Perform an epidural blood patch, injecting autologous blood into the epidural space at or near the puncture level to clot and seal the dural leak and restore cerebrospinal fluid pressure
  • E) Prescribe a long course of opioids and discharge the patient with no procedural intervention

ANSWER: D

Rationale:

The postural headache (mild supine, disabling upright) with neck stiffness and photophobia two days after a known dural puncture is a classic disabling post-dural puncture headache that has failed conservative care. The definitive treatment is an epidural blood patch: autologous blood injected into the epidural space at or near the puncture level clots and seals the dural leak, restoring cerebrospinal fluid pressure and typically relieving the headache promptly.

  • Option A: Option A prolongs ineffective conservative care while the patient cannot function or care for her newborn.
  • Option B: Option B treats an infection that is not indicated by this classic postural-headache picture.
  • Option C: Option C would remove more cerebrospinal fluid, worsening the low-pressure state driving the headache.
  • Option E: Option E manages symptoms with opioids while ignoring the available definitive intervention.

9. A patient with a thoracic epidural infusion for postoperative analgesia is noted to have mild new weakness in one leg. He is afebrile with no back pain. You stop the infusion and reassess 45 minutes later: the motor strength has substantially recovered and there is no further progression. What is the most appropriate interpretation and next step?

  • A) This is an epidural hematoma; proceed directly to emergent surgical decompression without imaging
  • B) This is an epidural abscess; begin antibiotics and continue the infusion
  • C) This is pharmacologic over-blockade, supported by recovery of strength once the infusion was stopped; resume analgesia at a lower concentration or rate (or a more dilute solution) and continue to monitor motor function
  • D) Increase the infusion rate to confirm the block is pharmacologic before making any other decision
  • E) Remove the catheter at once without regard to anticoagulant timing and discharge the patient

ANSWER: C

Rationale:

The motor-block workup hinges on the response to stopping the infusion. Weakness that substantially resolves and does not progress after the infusion is stopped, in an afebrile patient without back pain, is characteristic of pharmacologic over-blockade rather than a compressive lesion. The appropriate step is to resume analgesia at a lower concentration or rate (or a more dilute solution) and continue monitoring motor function; urgent imaging is reserved for weakness that persists or progresses despite stopping the infusion.

  • Option A: Option A leaps to emergent decompression for a deficit that has already reversed, which does not fit hematoma.
  • Option B: Option B misassigns an afebrile, painless, reversible deficit to abscess and would continue the offending infusion.
  • Option D: Option D would deepen the block and obscure assessment.
  • Option E: Option E removes the catheter without observing anticoagulant timing and discharges a patient who still requires monitoring.

10. A 79-year-old with severe aortic stenosis requires open reduction of a hip fracture. The cardiology consult emphasizes that this preload-dependent, fixed-output physiology tolerates only gradual hemodynamic change. You plan a neuraxial technique. Which approach best protects this patient's hemodynamics?

  • A) A full-dose single-shot spinal, because a single rapid block is simplest and avoids a catheter
  • B) A combined spinal-epidural technique using a deliberately low intrathecal dose supplemented through the epidural catheter in titrated increments, so the block level is reached gradually and the abrupt profound sympathectomy of a full single-shot spinal is avoided
  • C) A high-dose hyperbaric spinal placed with the patient head-down to guarantee a rapid high block
  • D) General anesthesia is mandatory because all neuraxial techniques are absolutely contraindicated in aortic stenosis
  • E) A rapid large-volume epidural bolus to establish a dense block as quickly as possible

ANSWER: B

Rationale:

In severe aortic stenosis the heart cannot compensate for an abrupt fall in afterload and preload, so the danger of neuraxial anesthesia is the speed and depth of the sympathectomy, not its use per se. A combined spinal-epidural with a low intrathecal dose, supplemented through the epidural catheter in titrated increments, builds the block gradually and avoids the rapid profound vasodilation and preload loss of a full single-shot spinal, matching the requirement for gradual hemodynamic change. Option A imposes exactly the abrupt sympathectomy this physiology tolerates worst. Option C deliberately drives a rapid high block, the most dangerous choice here. Option E produces a rapid dense block, again the opposite of the gradual approach required.

  • Option D: Option D overstates the contraindication; carefully titrated neuraxial technique can be used, and the goal is gradual onset.

11. A 52-kg patient with compensated cirrhosis undergoes outpatient rotator cuff repair and is to be discharged home with a continuous peripheral nerve block (CPNB) infusion via a portable pump for postoperative analgesia. Considering her low body weight and hepatic impairment, how should the infusion be set up and the patient counseled?

  • A) Use the standard infusion rate and provide no special instructions, since ambulatory CPNB carries no systemic risk
  • B) Choose bupivacaine at a high rate to ensure dense analgesia, because its narrow cardiac safety margin is advantageous over days
  • C) Increase the infusion rate to account for her small size, since small patients eliminate drug faster
  • D) Avoid any local anesthetic and send her home on oral opioids alone, since peripheral catheters always accumulate to toxic levels
  • E) Use a reduced infusion rate, preferably with ropivacaine for its wider cardiac safety margin, and counsel the patient and a caregiver to recognize early local anesthetic systemic toxicity (LAST) symptoms such as perioral numbness, tinnitus, and metallic taste, with fall precautions for motor block and clear instructions on whom to call

ANSWER: E

Rationale:

Over a multi-day ambulatory infusion, local anesthetic can accumulate, and both low body weight (smaller volume of distribution) and hepatic impairment (reduced metabolism) raise plasma concentrations for any given rate, narrowing the safety margin. The appropriate plan is a reduced infusion rate, ropivacaine for its wider cardiac safety margin, and explicit education of the patient and caregiver about early LAST symptoms, fall precautions from motor block, and whom to contact. Option A wrongly denies systemic risk and omits essential safety counseling. Option B selects the agent with the narrower safety margin at a high rate, misframing a hazard as a benefit. Option D needlessly discards effective regional analgesia and overstates the risk as universal toxicity rather than a manageable, rate-dependent concern.

  • Option C: Option C inverts the pharmacokinetics, since a small, hepatically impaired patient clears drug more slowly.